Can't seem to get rid of that belly fat? Scientists Discover: Probiotics Can Eat Away Visceral Fat!
01丨Be Careful of Visceral Fat Obesity
"I've lost 5 pounds, but my belly hasn't shrunk at all?"
"The health report says 'excessive visceral fat,' but I don't even look fat!"
If you have the same concerns, don't rush to run 10 kilometers or eat meal replacements—the latest international research tells us: maybe it's not that you're not working hard enough, but that you don't have enough "lean bacteria" in your gut.
Many people equate "fat" with the numbers on the scale, but ignore a more dangerous signal—visceral fat.
Obesity is divided into subcutaneous fat obesity and visceral fat obesity.
02丨Visceral Fat is an Invisible "Time Bomb"
Visceral fat hides deep in the abdominal cavity, surrounding the liver, pancreas, and intestines, even if your weight is normal. Excessive visceral fat can thicken blood vessel walls and reduce vascular elasticity, thereby increasing blood pressure and cholesterol levels, and increasing the probability of cardiovascular diseases. At the same time, it affects the secretion of insulin in the body, increasing insulin resistance, making the body more prone to high blood sugar.
Therefore, as long as visceral fat exceeds the standard, the risk of type 2 diabetes, fatty liver, and cardiovascular events will increase exponentially.
03丨Lean Bacteria Help "Eat Away
F3-2 metabolites downregulate fatty acid synthase (FAS), allowing the liver and fat cells to "store less oil"
Related Experiments
After intervention with Lactiplantibacillus plantarum F3-2, the morphology of mouse epididymal fat cells improved, and the cells became significantly smaller, with an increased number of small cells.
Experimental Conclusion:
Lactobacillus plantarum F3-2 reduces the volume of fat cells and alleviates excessive fat accumulation.
04丨Activate Fat Breakdown
Bifidobacterium animalis subsp.lactis F1-7
accelerates cholesterol breakdown through the FXR/FGF15 bile acid pathway; at the same time, it increases "good cholesterol" HDL and reduces "bad cholesterol" LDL
Related Experiments
Experimental Conclusion:
F1-7 can reduce the gene expression levels of intestinal farnesoid X receptor (FXR), fibroblast growth factor 15 (FGF15), and Niemann-Pick C1-like 1 (NPC1L1), and upregulate the gene expression level of liver X receptor (LXR). F1-7 regulates bile acid metabolism by reducing FXR expression and participates in the FXR/FGF15 pathway to promote cholesterol breakdown, effectively improving hyperlipidemia induced by high-fat diet in mice.
Related Experiments
Experimental Conclusion:
F1-7 can effectively reduce the levels of serum triglycerides (TG), total cholesterol (TC), and low-density lipoprotein cholesterol (LDL) in mice on a high-fat diet, and increase the level of high-density lipoprotein cholesterol (HDL).